Deans' stroke musings: Relation of the Bilateral Earlobe Crease to Endothelial Dysfunction

Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, April 8, 2017

Relation of the Bilateral Earlobe Crease to Endothelial Dysfunction

You might want to know how your doctor is treating your earlobe crease. What protocol is being used?
http://www.ajconline.org/article/S0002-9149%2817%2930353-3/fulltext?rss=yes

Nozomu Oda, MD

Tatsuya Maruhashi, MD

Shinji Kishimoto, MD

Masato Kajikawa, MD

Yumiko Iwamoto, MD

Akimichi Iwamoto, MD

Shogo Matsui, MD

Takayuki Hidaka, MD

Yasuki Kihara, MD

Kazuaki Chayama, MD

Chikara Goto, PhD

Yoshiki Aibara, MS

Ayumu Nakashima, MD

Kensuke Noma, MD

Harutoyo Hirano, PhD

Yukihito Higashi, MD

Correspondence information about the author MD Yukihito Higashi

Email the author MD Yukihito Higashi

DOI: http://dx.doi.org/10.1016/j.amjcard.2017.03.029

Article Info

Abstract

The presence of an earlobe crease (ELC) may be a simple sign to predict atherosclerosis. We evaluated the relationship between ELC and vascular function. We measured flow-mediated vasodilation (FMD) and nitroglycerine-induced vasodilation (NID) and observed bilateral earlobes in 400 consecutive subjects. At first, the subjects were divided into 3 groups: non-ELC group, unilateral ELC group and bilateral ELC group. FMD and NID were significantly lower in the unilateral and bilateral ELC groups than in the non-ELC group. After adjustment of cardiovascular risk factors, bilateral ELC, but not unilateral ELC, was associated with lower FMD and lower NID. We also investigated whether an increase in the number of ELCs worsens endothelial function, whether the difference in ELC structure (cross stripes and/or ramification) affects endothelial function, and whether endothelial function is impaired in subjects with superficial wrinkles depending on age. Number of ELCs, shape of the ELC, and superficial wrinkles were not associated with endothelial dysfunction. In conclusion, these findings suggest that the presence of bilateral ELCs is associated with vascular dysfunction.